<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Jonathan Wolpaw</style></author><author><style face="normal" font="default" size="100%">Seegal, R. F.</style></author><author><style face="normal" font="default" size="100%">Dowman, R.</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Chronic exposure of primates to 60-Hz electric and magnetic fields: I. Exposure system and measurements of general health and performance.</style></title><secondary-title><style face="normal" font="default" size="100%">Bioelectromagnetics</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">60-Hz fields</style></keyword><keyword><style  face="normal" font="default" size="100%">central nervous system</style></keyword><keyword><style  face="normal" font="default" size="100%">electric field</style></keyword><keyword><style  face="normal" font="default" size="100%">magnetic field</style></keyword><keyword><style  face="normal" font="default" size="100%">primate</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">1989</style></year><pub-dates><date><style  face="normal" font="default" size="100%">01/1989</style></date></pub-dates></dates><urls><web-urls><url><style face="normal" font="default" size="100%">http://www.ncbi.nlm.nih.gov/pubmed/2751702</style></url></web-urls></urls><volume><style face="normal" font="default" size="100%">10</style></volume><pages><style face="normal" font="default" size="100%">277–288</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">We exposed pigtailed macaques (Macaca nemestrina) to electric (E) and magnetic (B) fields at strengths of 3 kV/m and 0.1 G, 10 kV/m and 0.3 G, and 30 kV/m and 0.9 G for three 21 day segments. These three exposure segments were preceded and followed by 21 day sham exposure segments. Additional animals received only sham exposure for five 21 day segments. Detailed description of the exposure chamber and field generation apparatus is given. We evaluated measures of animal well-being, including weight, blood chemistry, blood cell counts, and performance on a simple motor task, and performed postmortem examinations. Reliable and consistent results were obtained throughout data collection. None of the measures evaluated was significantly affected by E- and B-field exposures. Data obtained during actual exposure segments were not distinguishable from those obtained during the initial and final sham exposure segments, nor were they different from data obtained from the sham-exposed animals. Thus, field exposure had no apparent effects on general health or performance.</style></abstract></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Jonathan Wolpaw</style></author><author><style face="normal" font="default" size="100%">Dowman, R.</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Spinal stretch reflex and cortical evoked potential amplitudes versus muscle stretch amplitude in the monkey arm.</style></title><secondary-title><style face="normal" font="default" size="100%">Electroencephalography and clinical neurophysiology</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">muscle stretch</style></keyword><keyword><style  face="normal" font="default" size="100%">primate</style></keyword><keyword><style  face="normal" font="default" size="100%">Somatosensory Cortex</style></keyword><keyword><style  face="normal" font="default" size="100%">somatosensory evoked potential</style></keyword><keyword><style  face="normal" font="default" size="100%">spinal reflex</style></keyword><keyword><style  face="normal" font="default" size="100%">stretch reflex</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">1988</style></year><pub-dates><date><style  face="normal" font="default" size="100%">04/1988</style></date></pub-dates></dates><urls><web-urls><url><style face="normal" font="default" size="100%">http://www.ncbi.nlm.nih.gov/pubmed/2450738</style></url></web-urls></urls><volume><style face="normal" font="default" size="100%">69</style></volume><pages><style face="normal" font="default" size="100%">394–397</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">While investigating operant conditioning of the primate spinal stretch reflex (SSR), we studied SSR amplitude and cortical somatosensory evoked potential (SEP) amplitude as stretch amplitude changed in the monkey arm. Initial muscle length and background EMG activity remained constant. With change in stretch amplitude (and proportional change in stretch velocity and acceleration), changes in SSR and SEP amplitudes were respectively 0.75 and 0.66 as great. The lesser change in SSR amplitude may reflect saturation of Ia afferents, while that in SEP amplitude may also reflect participation of other peripheral receptors.</style></abstract></record></records></xml>